Alzheimer’s

The results have important implications for the design of microglia-centered therapeutics for AD.

Alleviating brain aging and COVID-19 pathology with senolytic therapy.

In vivo microhemorrhages linked to erythrocyte-brain endothelial interactions.

Inhibition of GCPII lowers pT217Tau in the brains of aged macaques.

Early mitochondrial hypermetabolism in Alzheimer's mice.

BMAL1 loss in oligodendroglia impacts myelination and sleep.

Uncovering undiagnosed cognitive impairment in the U.S. Medicare population.

Unveiling the peroxisomal Pex1/Pex6 ATPase complex with substrate.

MKK6-p38α complex: Defining MAPK activation.

Stem-ing the tide of Alzheimer’s.

Study highlights microglia degeneration in brain caused by iron toxicity.

Study examines the use of monoclonal antibody therapies for treating Alzheimer disease (AD).